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How can Persistent Sexual Arousal Symptoms or orgasms happen?? Any inflammation or swelling phenomena in or against the prostate, penis, seminal vesicles, urethra, clitoris, vagina, cervix and uterus can result in sexual urgency, PSAS or continuous orgasms. In addition to cyst, fibroid, tumor or cancer outgrowth, excessive Prostaglandin E2 can directly stimulate the prostate, penis, seminal vesicles, urethra, clitoris, vagina, cervix and uterus for more sex and let you want to have orgasm or to ejaculate when your pituitary don't release enough prolactin to suppress sexual and orgasm urgency. Excessive prostaglandin E2 and oxytocin down there will keep your bottom inflaming, very wet and very sensitive all day long and tickle your orgasm responses (premature orgasms) any times. In fact, excessive prostaglandin E2 is responsible for cyst, fibroid, tumor or cancer outgrowth too. But, you need some prostaglandin E2 balanced by prostaglandin E1 and E3 to keep you body and bone health. OK, here are the facts to help you understand this PSAS disorders: Fact 1: Today, many dairy producers are using analog prostaglandin E2 to induce cows to come into heat for milking release. Similarly, we human beings can get the similar responses too, particularly taking non-organic dairy products that contain residual prostaglandin E2 analog. Fact 2: Prostaglandin E2 and oxytocin initiate orgasm and labor uterine contraction. In turn, orgasm and labor induce more Prostaglandin E2 release, but orgasm can trigger the pituitary to release prolactin to prolong refraction time for the next run of sex, and shot down sexual desire and erection for both sexes. Prostaglandin E2 and oxytocin heighten your sexual arousal and orgasm responses, but prolactin suppress and dampen both. You need both oxytocin and prolactin in your bloodstream to feel sexually being satisfied at the end of sex, but masturbation won't induce much oxytocin release. However, masturbation can give you a lot of prostaglandin E2 in your clitoris, penis, urethra, vagina and prostate, and trigger your adrenal medulla and hypothalamus to release a lot of epinephrine into your bloodstream, resulted in hot blood and in persistent sexual arousal after masturbating. Fact 3: Women/Men or animal in heat. When men, women and animals are in heat, their penis, prostate, clitoris, vulva and pelvic cavity are frequently swelling or erecting without pains and become extremely sensitized by Prostaglandin E1, E2 and E3, oxytocin, testosterone, DHT, Nitric Oxide, as described in http://www.actionlove.com/extra/animal.htm. During the the in-heat state, they always want to mate or be mated anytime. Once the female become pregnant, the fertilized-egg implantation and the placenta release progesterone and estrogen to trigger the pituitary to release prolactin and to stimulate the liver to release SHGB protein to freeze testosterone and DHT in the bloodstream. Therefore, the female heat (Sexual Arousal) becomes subsided. But men won't change until their pituitary-testicular axis is fully exhausted for a constantly elevation of prolactin. However, an elevation of prolactin may not stop over-masturbation or sex practices if a high level of inflammatory hormone prostaglandin E2 can continuously sensitize the sexual nerves in the clitoris, penis, G-spot, vagina, urethra and prostate, even if they experience erectile dysfunction. That is, they become addictive to sex or masturbation. Fact 4: Pregnant Woman Sexual Responses. The pregnant women's clitoris, vulva, uterus, cervix and pelvic cavity are swollen and enlarged without pains by Prostaglandin E1, E2 and E3, oxytocin, Nitric Oxide, and estrogen. If the placenta releases excessive estrogen and progesterone and the liver and pituitary continuously releases more and more SHGB and prolactin, respectively, in response to the continuous elevation of estrogen and progesterone, the pregnancy will reduce or shut down sexual arousal or orgasm responses. This means the swelling of the clitoris, vulva, uterus, cervix and pelvic cavity may not increase, but kill, sexual arousal or orgasm ability. If the liver won't release sufficient SHGB protein so that the pituitary releases a high level of oxytocin with a low level of prolactin, pregnancy will boost sexual desire and orgasm responses, maybe leading to miscarriage. Oral, implant or injection birth controls use artificial progesterone and estrogen to mimics the pregnancy condition to shut down the hypothalamus-pituitary-ovarian axis, to overload (poison) the liver P450 detoxification system for excessive SHGB protein release and for reduction of neurotransmitters dopamine, acetylcholine and serotonin release, to interfere with the thyroid function, and to reduce prostaglandins synthesis. This is why the birth controls chemically castrate the female for no libido, sex and orgasm. When the liver fails to detoxify all the artificial progesterone and estrogen into natural ones, women on the birth controls can experience severe physiological and psychological disorders, vaginal discharge, clitoral and G-spot desensitization and erectile dysfunction, frigidity (lack of oxytocin and testosterone / DHT). Some unfortunately women get a permanent damage in the hypothalamus-pituitary and some experiences premature menopause after they get off birth control, as described in http://www.actionlove.com/extra/contracept.htm , http://www.actionlove.com/extra/shot.htm , and http://www.actionlove.com/extra/discharge.htm Fact 5: Clitoral, Vulva, vaginal, uterine, prostate, urethral and Pelvic Inflammation - Persistent sexual arousal is an inflammatory responses to chronic release of epinephrine and prostaglandin E2 with excessive oxytocin, testosterone and DHT release. This condition can happen while women get off birth control while their Clitoral, Vulva, vagina and Pelvic tissues were inflamed by chronic over-masturbation induced excessive prostaglandin E2 release. Here, chronic over-masturbation keeps the epinephrine level high and constantly induce excessive inflammatory hormone prostaglandin E2, without prostaglandins E1/E3 and nitric oxide, into the bloodstream for repair of the masturbation-damaged nerves, tissues and blood vessels. After women get off birth control or turn to 30-something or 40-something, the liver and hypothalamus-pituitary-ovarian systems have been luckily and fortunately unlocked for more oxytocin, testosterone and DHT release, but less prolactin and liver SHBG protein which may become too low to suppress or counter the powerful effects of prostaglandin E2, oxytocin, testosterone and DHT on sexual arousal and orgasm. Young men, with excessive prostaglandin E2, oxytocin, DHEA, testosterone and DHT in the bloodstream are likely to experience persistent sexual arousal for excessive sex and masturbation. Therefore, Persistent sexual arousal symptoms includes inflammatory clitoris, vulva, G-spot, uterus, cervix, penis, urethra, prostate and bladder for sexual arousal, clitoral pains, pelvic pains (interstitial cystitis or IC), prostate pains, bladder pains, urethral pains, penile pains, testicles pains (testicular Pains, joint pains, stomach pains, headaches, dizziness, vertigo, frequent urination urgency, vaginal fluid leakage, semen/precum leakage, unwanted ( spontaneous ) orgasms, fatigue, exhaustion, and other body pains and cramps. With a strong action of prostaglandin E-2, oxytocin and epinephrine (for sympathetic nervous Fight responses), internal pelvic sympathetic nerves will frequently produce uterine or prostate contraction for persistent orgasm and fluid leakage. Solution for Persistent Sexual Arousal Syndrome and interstitial cystitis induced by COX-2 over-expression and inflammatory hormone prostaglandin E-2 in the pelvic cavity tissue and for control of unwanted, spontaneous sexual orgasm |
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